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Author Topic: Managing Hog Health  (Read 2001 times)
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mikey
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« on: September 29, 2008, 03:12:35 AM »

Anatomy, Physiology and Technical Terms
(15) An understanding of disease processes can be difficult if the reader has little scientific background. For those who have not had training in biological subjects this chapter looks at the basic areas of knowledge necessary to appreciate and understand the information given in this book. All the diseases and conditions mentioned briefly in this chapter are discussed in detail later.

The anatomy and physiology of the pig can be broadly grouped into eleven inter-related systems:

Circulatory system.
Digestive system.
Endocrine system.
Immune system.
Muscular system.
Nervous system.
Reproductive system (Male and Female)
Respiratory system.
Sensory systems.
Skeletal system.
Urinary system.
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« Reply #1 on: September 29, 2008, 03:15:41 AM »

Definition of Health and Disease
(30) The term "Health" means different things to different people. Absence of diseases is clearly a prerequisite but in pigs it means more than that. It is a state of physical and psychological well-beings that allows the pig to express its genetic potential for maximising productivity, reproductive performance and lean meat production.
The term "disease" means an unhealthy disorder of body and mind, sometimes with pain and unease, that is likely to prevent the pig from exploiting its genetic potential resulting in lowered productivity.

The level of clinical disease is described by the term morbidity. Disease can be clinical (i.e. the affected pig shows clinical signs) or sub-clinical (the affected pig shows no obvious clinical signs). Sub-clinical disease can also have an adverse affect on productivity. You should distinguish between sub-clinical disease and sub-clinical infection,. Every healthy herd, without exception, carries a multitude of potentially pathogenic infections, mainly in the gut but also in the nose, throat, skin and genitals, which are not causing disease either clinical or sub-clinical.

There is a delicate balance between these potential pathogens and the pigs' immunity to them. Any physical or psychological disturbance of this immunity may render the pig susceptible.

Good husbandry, including good stockmanship aims to avoid such disturbances, provided the more virulent diseases are absent (e.g. atrophic rhinitis, TGE). Good husbandry means good housing, good nutrition and good management. Good stockmanship means care and attention to the pig's health and welfare.

This delicate balance between potential pathogens and the pigs immunity becomes even more precarious on a herd basis. By causing disease in small groups of pigs as a result of poor husbandry, the pathogenic organism multiplies up to a concentration that may overcome the more resistant pigs. The concentration again builds up and threatens to overwhelm the collective immunity of the herd (i.e. herd immunity). (Fig.2-0)



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mikey
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« Reply #2 on: September 29, 2008, 03:18:44 AM »

An introduction to managing health and disease
(60) There are many factors that affect the economic viability of modern day pig production. For a business to be successful it must take account of all of them. Disease is one that plays a significant role. The important factors that you might be able to control and improve are highlighted here. You should find it of value to consider each and assess them in relation to your own pig farm.
There are other factors, such as government regulations which are completely out of your control and so are omitted from this check list. Likewise, money available to the consumer is a national problem, rather than one at farm level. The important criteria on the farm, however, are the price of feed and the efficiency with which this is converted into liveweight gain and ultimately lean meat. The interaction between people, management, the environment and disease, dictates the efficiency of these conversions.

Factors that affect the economic viability of modern day pig production

Application of new technology.
Continuing education / training.
Control of pollution.
Daily liveweight gain.
Disease control and treatment.
Efficiency of feed conversion to lean meat.
Efficiency of pen utilisation.
Environmental restrictions.
Equipment cost and quality.
Genetic potential of the pigs.
Its control and treatment.
Labour cost.
Management and motivation of people.
Management decisions.
Market outlets.
Methods of feeding.
Number of pigs sold per year.
Nutrition and quality of the feed.
Planning.
Price of feed.
Reproductive performance.
Size of the herd.
Slaughter price.
Stocking densities.
The control of pollution.
Transport cost.
Use of records.
Weight and grade of pigs sold at slaughter.
Welfare requirements.
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« Reply #3 on: September 29, 2008, 03:23:48 AM »

Imunity - How the Pig Responds to Infection
(61) Infection is considered to have taken place when a virus, bacterium or parasite enters a pig and starts to multiply. If it is a potentially pathogenic organism it may change the normal structure and function of the pig. A study of these changes is called pathology and organisms causing disease are described as pathogens. Fig.3-2 shows the sequence of events that may take place when an infectious pathogen infects the pig.
 

Infection creates two scenarios; the first is when there is no disease. The immune mechanism of the pig responds to challenge and the infectious agent is either eliminated or remains within the body in a carrier state. The carrier pig may or may not shed the organism or may shed it intermittently. The second scenario is that of disease which is followed by an immune response and complete recovery and elimination of the pathogen or recovery with a carrier state, or death. Whether the organism causes disease or not is dependent on how virulent (the capability of the organism to produce disease) it is, how many organisms are present, what other concurrent infections are present, what protective mechanisms are available in the pig to prevent disease and what environmental or other factors are present that will lower the pigs immunity. For example, certain strains of Actinobacillus pleuropneumoniae are only mildly pathogenic and do not normally produce disease. However if they are in combination with PRRS virus the presence of both could produce severe pneumonia. A non pregnant gilt exposed to parvovirus infection only develops an immune response but no signs of disease because disease only occurs in the developing foetus. Nevertheless the gilt would become serologically positive in laboratory tests.

A successful and healthy pig farm is one in balance and harmony with the organisms that are present both in the environment and the pig. It is the manipulation of this balance that is so important to health and disease and ultimately to productivity and profitability.

It follows that if the numbers of organisms in the environment can be maintained at a minimum then the threshold level of organisms necessary to produce disease is unlikely to be reached. Examples here would be the excellent growth rates that can be achieved with all-in all-out systems in nurseries or flat decks, when pigs are housed on weld mesh or slatted floors and they are divorced from their faeces and potential enteric organisms. Likewise in respiratory diseases, the more pigs there are in a common air space, and the smaller the cubic capacity of that air space relative to the numbers of pigs, then the greater will be the numbers of aerosol organisms and the more severe disease.

To be a successful pig producer you should have some idea of how pigs resist infectious disease. Unfortunately, it is a complex subject and although here an attempt has been made to simplify it, it will probably still seem a little complicated.

The technical terms used in immunology (i.e. the study of immunity) are a major part of the problem. If you have not had a good grounding in biology they may be like a foreign language. If that is the case, or even if it is not, start by reading through the terminology below.

Terminology

Adjuvant - A substance added to an inactivated vaccine to make it more effective.

Antibodies - Complex large proteins (called gamma-globulins) which are produced by specialised cells in response to invading antigens and which stick specifically to the invading antigen neutralising it or triggering off a destructive reaction.
Antigen - Foreign invading substance (i.e. a substance which is not normally part of the pig's body), usually consisting of protein or part of a protein, which stimulates the body to produce antibodies. Antigens exist on the surfaces of bacteria, viruses and parasites.

Remember:

Expose your pigs to too many pathogenic organisms and you have sick pigs - poor performers.
Lower the level of pathogens in the environment and you have healthy pigs - high performers.
Antiserum - Serum with high antibody levels against a specific infection. It has usually been produced experimentally in laboratory animals by injecting the infection into them.
Blood sample - Whole blood sample taken hygienically with a syringe into a bottle or by a pin prick through the skin absorbing the droplet of blood with blotting paper.
Commensal bacteria - Bacteria that live permanently in or on the body without causing disease.
Epithelium - Cellular membrane (e.g. mucous membranes) containing epithelial and other cells.
Humoral immunity - Blood-borne immunity.
Hyperimmune antiserum - The same as antiserum above but emphasising its high titre.
Lymphocytes - Specialised defence cells in lymph nodes, other lymphatic tissue and the blood which produce antibodies or take part in cellular immunity.
Mucous membranes - Cellular membranes (e.g. those lining the gut) which secrete a sticky substance called mucus on to their surfaces.
Mucus - A clear sticky semi-liquid secreted by cells in mucous membranes.
Pathogenic infection - An infectious organism which has the potential to cause disease. This is in contrast to the many organisms that live normally in or on the body which never cause disease and are called commensals.
Phagocytes - Cells of the body whose special task is to engulf bacteria, viruses, or parasites in an attempt to destroy them. They are also called macrophages.
Phagocytosis - The process whereby the specialised cells of the body engulf bacteria, viruses or parasites in an attempt to destroy them.
Plasma sample - A whole blood sample taken hygienically with a syringe and mixed with an anti-clotting agent so that it remains liquid. The sample is spun fast in a centrifuge and the red and white blood cells sediment to a firm pellet at the bottom leaving a clear liquid - the plasma.
Serology - Tests done in the laboratory to detect the level of specific antibodies in serum samples ("ology" means study of - so literally serology means "study of serum").
Serum sample - A whole blood sample taken hygienically with a syringe and allowed to clot. The serum is the clear straw-coloured liquid which can be drawn of with a pipette. It contains the antibodies.
Titre - The concentration of a specific antibody in a serum sample. It is expressed as the amount by which the serum has to be diluted before a serological test goes negative.
Virulence - How pathogenic an organism is. Organisms with a high capability of causing disease are called highly virulent.

The main components that make up the resistance of a pig to infection may also seem complicated but if you refer to Fig.3-3 as you read it should also help you to understand the text better. You do not need to understand all the components but take note of the following:

What antibodies are.
What stimulates them to be produced.
The importance of colostrum and milk in providing immunity.
Why blood tests are done and how they are interpreted.
How vaccines work.



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« Reply #4 on: September 29, 2008, 03:27:39 AM »

The Breeding Female
(160) Problems associated with reproductive failure are often complex. In trying to resolve such problems it is essential to understand the important factors that maximise biological efficiency.
Key points to maximising reproductive performance


Use the breed of female that is suitable for your system.
Ensure that the breeding female exhibits good hybrid vigour.
Monitor the age of the sow and her continuing performance.
Collect the required records and use them to understand the problem.
Ensure an even parity spread across the herd. Litters 3-5 are most productive.
Use high levels of feed energy, protein and lysine in lactation and maximise feed intake.
Provide an equitable environment with even temperatures. Cleanliness is essential.
Compare the reproductive performance to the lactation length.
Maintain good body condition throughout pregnancy.
Have a sufficient number of boars available so that they need only be used once every 24 hours at most.
Maintain sow health and immunity to disease.
Assess the age of the gilt at first service, relative to life time performance in the herd.
Assess the effectiveness of sow management from weaning to 21 days post-service.
Make sure that the right type of stockperson is in charge of mating. The ability of the stockperson has a direct effect on reproductive performance
Most of the major breeding (seedstock) companies now develop lines rather than standard breeds. The lines are generally based on breeds but have been developed differently. Thus one line A may be based predominantly on the Large White breed but has been developed as a dam line by selecting for prolificacy (No. of piglets born alive per litter and No. of litters born per year) and mothering ability (No. of piglets weaned per sow per year). Line D may also be based on the Large White breed but has been developed as a sire line by selecting primarily for growth rate, feed conversion efficiency and lean meat. This sire line development is often done at the expense of prolificacy and so if you are a commercial producer you should never select female back-crosses sired by such boars for breeding.
Efficient reproduction starts with females of high genetic potential for prolificacy and this includes good hybrid vigour (heterosis). The hybrid female is the progeny of the male of one breed or dam line (A), crossed with the female of another breed or dam line (B) to produce the commercial F1 gilt (AB). This animal is usually highly prolific in terms of pigs produced per sow per year. Some breeding companies cross the F1 with a third breed or dam line (C) to produce their commercial hybrid or three way cross. Such pigs are designated by a variety of numbers and names and Fig.5-1, illustrates the differences that may occur between (ABxC) crosses depending on the level of hybrid vigour. Pure breeding produces the worst scenario, hence the reason why, at a commercial level no farmers should use pure bred females and boars of the same breed. In the past it has been common practice, commercially, to breed back from the F1 by using a boar of the same breed to produce a further breeding female that is 3/4 pure breed (A),1/4 alternate breed (B). However, there is invariably loss of hybrid vigour and even in farms that are efficient, performance is usually less efficient than the F1.





Fig.5-2 lists the essential features of different breeding combinations and it is important when selecting the source of your gilts to remember that there can be a considerable difference in the prolificacy of the dam lines and in the expression of hybrid vigour between one breeding company and another.





The age of the sow also plays an important part in reproductive efficiency. In Fig.5-3 you will see that the best reproductive performance is in litters 3, 4 and 5. Although by litter 8 the total number of pigs born may be greater there is often wide variation in the size of individual pigs at birth, an increasing number being too small or runts. There is also an increase in the numbers of stillborn with a reduction in live births. Furthermore, with age sows tend to be more clumsy and lazy, with higher levels of pigs laid on and an increased mortality, often 3% or more above those of the efficient parities. Sows in litters 3 to 5 should rear at least 24 pigs per annum but by the 8th litter and above this often drops below 21. Levels of reproductive efficiency should also be compared to those being achieved by the incoming gilts. Culling decisions should be made in light of the performance of your first litter gilts, the maintenance of the mating programme and the availability of older sows as foster mothers.

The parity distribution in the herd necessary to maximise production is shown in Fig.5-4.





Sow management from the day of weaning to 21 days post-service influences reproductive efficiency. If you have a problem, this is the first area that should be assessed.

Consider the overall plan in Fig.5-5 which defines the critical parameters necessary to maximise reproductive efficiency.





The maintenance of gilt and sow numbers is the first critical factor. There must always be sufficient females available to maintain a mating programme.

A shortfall in numbers available is the most common failure on most pig farms.

From point of mating to point of farrowing there can be a variety of pregnancy failures which are dealt with later in this chapter. For every 100 sows served in a given period of time less than 100 will farrow. This in percentage terms is described as the farrowing rate. Failures of animals to farrow are associated with repeat matings, abortions, sows not in-pig, deaths, culls and/or disease.

Farrowing Rate =

Nos. of sows farrowed x 100
Nos. of sows served

How to approach a problem on the farm
Of all the problems on the pig farm, understanding and resolving a reproductive problem can be the most challenging. However, if a logical approach is taken, the causes of the reproductive failures can usually be worked out and corrective actions taken. The approach outlined in Fig.5-6 relates the performance of the herd to accepted levels of efficiency, this then defines the area of failure and finally the problem is identified. Further investigations are then necessary to understand what has precipitated the problem and these are carried out by examining the management procedures, records and performing appropriate pathological tests. An important objective here is to determine if the problem is of infectious or non infectious origin or a combination of both. The end results are either farrowing rate loss, a reduced litter size, anoestrus or any combination.



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« Reply #5 on: September 29, 2008, 03:31:11 AM »

Diseases Affecting Reproduction
(200) The previous chapter has reviewed the various management and environmental factors that have an adverse affect on reproductive efficiency. The description of the diseases here concentrates on their effects on reproduction but in some of them infertility is only part of a much broader picture. In such cases references to specific chapters are given. There is often an overlap between infectious and non infectious infertility and in many cases the two are interrelated. It is important to determine if there is an infectious component to a problem in a herd because corrective measures may involve both treatment and management procedures. Fig.6-1 demonstrates a pathway that can be used to identify the causes of an infectious infertility problem. It asks the question what diseases are present in the herd, because any one of these may have an occasional effect. Such diseases are listed in Fig.6-2 and Fig.6-3. For example, if a herd is infected long term with aujeszky's disease virus, porcine parvovirus (PPV), porcine reproductive and respiratory syndrome (PRRS) or swine influenza (SI) then at varying intervals following the initial herd outbreak there may be reproductive failures, albeit in many cases at individual sow levels. If a herd is free from these infections and then become infected with one of them an acute episode of that disease will occur. This will be manifest by reproductive failure including increased numbers of abortions, repeats on a normal and abnormal cycle and in the case of those viruses that cross the placenta, there will be foetal death, mummified and stillborn piglets.
Diseases such as porcine parvovirus (PPV), PRRS and leptospirosis may infect the sow without causing other clinical signs. Usually in such cases the disease picture is sporadic (unless it appears for the first time) and a detailed examination of records helps to clarify this.

There are nine main viruses that can cause reproductive disease, but only five of these are really important in those countries where they occur. The five are:

Aujeszky's disease (AD) virus or pseudorabies virus (PRV).
Porcine parvovirus (PPV).
Porcine reproductive and respiratory syndrome virus (PRRS).
Classical swine fever virus (CSF).
Swine influenza virus (SI).
Bovine viral diarrhoea virus (BVDV) border disease virus (BDV) and enterovirus (SMEDI) may cause sporadic reproductive problems but in most countries disease would be uncommon. Encephalomyocarditis virus (EMCV) varies in its pathogenicity in different regions of the world. In Europe it is rarely implicated in porcine disease, in the Caribbean it causes heart problems but in North America it is associated with reproductive problems
Most bacteria are opportunist invaders and usually only affect a few individual breeding females, a good example is erysipelas. The exceptions are leptospirosis and brucellosis which are herd problems.

Some of the clinical signs and pathological effects that viral and bacterial agents have on fertility are shown in Fig.6-2 and Fig.6-3.






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« Reply #6 on: September 29, 2008, 03:34:43 AM »

Signs of Disease
(230) During the dry period diseases may appear in individual cases or in outbreaks involving a number of animals. Infectious diseases may attack more than one system of the body and the clinical signs then relate to the failure of that particular system and include the following:
The digestive system

Anaemia.
Blood or mucus in faeces.
Colic
Change in the faecal consistency - constipation, diarrhoea.
Dehydration.
Distension of the abdomen.
Inappetence.
Salivation.
Variable body condition.
Vomiting.
The locomotor system
Abscesses.
Fractured bones.
Incoordination.
Lameness.
Paralysis.
Swellings of joints, muscles and tendons.
Trembling
The nervous system
Blindness.
Fits and convulsions.
Loss of balance, middle ear infections.
Loss of leg function.
Muscular tremors.
Nystagmus (jerky eye movements), meningitis.
Paraplegia / paralysis.
The respiratory system
Coughing.
Discharges from the eye.
Discharges from the nose.
Heavy breathing - pneumonia.
Sneezing.
The skin
Abscesses.
Cuts and bruises.
Dermatitis (inflammation).
Discoloration.
Erysipelas lesions. Diamond markings.
Excessive hair growth.
Fly bites.
Greasy skin.
Haemorrhage.
Jaundice
Lice.
Mange. Scratching.
Paleness - anaemia.
Rodent bites.
The urogenital system
Abnormal discharges from the vulva.
Abnormal oestrus
Abnormal urine colour.
Abortion.
Blood, mucous, or pus in the urine.
Fevers.
Mucus indicative of an early abortion.
Oestrus, anoestrus.
Pregnancy failures.
Wet urine-stained vulva.
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« Reply #7 on: September 29, 2008, 03:44:28 AM »

Achieve Low Pre-Weaning Mortality
Note: Indoor production unless otherwise stated

(300) An efficient and well managed farrowing house should be able to achieve an average pre-weaning mortality of between 5 and 8%. One objective of this chapter is to discuss those key factors that will achieve such targets. Fig.8-1 identifies the major components of the management system that are relevant to low pre-weaning mortality. A good recording system to identify the problem area or areas of failure is a prerequisite.




Good planning

The planning and control of the mating programme, which ultimately dictates the farrowing programme, are important for disease control because they determine the rate at which pigs move in and out of the farrowing rooms and indeed the whole of the farm. The quality and breeding of the dam and its health status also contribute significantly towards reducing piglet mortality, but the greatest influence arises from management techniques that are adopted around farrowing and during the first 72 hours after birth. A healthy viable piglet with a good birth weight must interact with a good farrowing house environment. Maximising the numbers of piglets reared per breeding female per annum is a major contributing factor to ultimate economic viability of the unit. "Without pigs you can not sell pig meat" is an obvious but little appreciated statement. Fig.8-2 shows the effect on cash flow by increasing the pigs per sow per year from 18 to 25 and most of this economic output will be net profit because the overheads of the unit are not increased by any significant amount.





Pigs sold per sow per year multiplied by the weight of pig leaving the farm determines the kilograms of meat sold per annum and economic output. It is important to maximise this by achieving the maximum biological efficiency of the breeding female on the farm. However over production initiated by too many matings in a given period of time, can have a depressant effect on both health and the efficient use of feed and a study of Fig.8-3 highlights the progression of events that can take place. (See also chapter 3 Management of mating). Note the hidden effects that over use of boars and excessive numbers of matings may have on disease throughout the whole of the production system.



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« Reply #8 on: September 29, 2008, 03:48:53 AM »

Maximum Productivity
(400) The production of healthy weaners is a complex interaction between disease, the environment and management. Management decisions and procedures however are the initiating factors that cause abnormal conditions in the immediate post-weaning phase. These factors include:
The production and maintenance of a healthy sucking pig.
An adequate weaning weight for the weaning system.
An optimum age of pig for the weaning system.
An environment with the necessary temperature, ventilation and humidity for the age and weight of the pig.
A correct diet for the age of the pig.
Good feeding procedures.
An all-in all-out system.
Key points to producing a healthy sucking pig
Use a hybrid or cross bred female.
Satisfy the nutritional requirements of the sow both in pregnancy and lactation.
Bring a healthy sow into the farrowing house.
Achieve good birth weights.
Ensure each pig receives maximum colostrum at birth.
Use a high energy and lysine diet in lactation to provide maximum nutrition for the litter.
Provide good farrowing house management and hygiene. (Chapter Cool.
Vaccinate the gilt and sow against E. coli diarrhoea.
Maintain an even creep temperature.
Provide fresh uncontaminated creep feed pre-weaning.
Age and weight of the pig at weaning
Successful weaning requires a combination of both minimum age and weight at the time of weaning to suit the weaning system. Weaning ages generally range from 14 to 28 days with most intensive farms having a mean between 21 and 26. As weaning age is reduced it is important to appreciate the potential effects this might have, not only on the pig but also on the sow.

The effects of reducing weaning age

The younger the pig the poorer its appetite at weaning.
Poor feed intake results in lower daily liveweight gain.
The younger the pig is weaned the less efficiently it will adapt to and digest solid food.
Highly specialised diets are required.
More weaning accommodation is required.
A more exacting weaning environment is required; more supplementary heat, more labour, more costly housing and higher creep costs.
The piglet is more susceptible to enteric diseases.
There is often an increase in post-weaning mortality.
A shorter lactation may reduce subsequent litter sizes and conception rates.
The days from weaning to first mating interval may be increased with more sows showing vulval discharges and found not pregnant.
Pigs produced per sow per annum or pigs weaned per crate can be increased.
Pigs may be healthier if weaned away from the farm (SEW).
The question is "How do you determine the best age at which to wean on your farm"?
This is dependent on:
The number of farrowing crates.
The milking capabilities of the sows, the breed and reproductive efficiency.
Litter size.
A good weight for age at weaning.
The health of the piglets at weaning time.
The quality and digestibility of the creep feed.
Weaning accommodation that will satisfy the pig's requirements.
A weaning age that results two weeks later in a healthy weaner which has achieved maximum daily liveweight gain. (Fig.9-1).
A weaning age that does not depress efficient reproductive performance in the sow.
The use of segregated early weaning techniques to improve health status.



Feed intake after weaning
Optimum levels of feed intake post-weaning maximise daily gain and feed conversion efficiency and reduce costs per kilogram of liveweight gain and energy requirements.

The heavier the pig at weaning the more efficient is the growth and feed conversion during the next four weeks and the quicker it reaches slaughter weight.

Changes in the intestine of the pig at weaning
Fig.9-2 shows the cross section of the small intestine of the weaned piglet to consist of many thousands of finger like projections called villi, which increase the absorptive capacity of the small intestine. During suckling they are continuously bathed by sows milk which contains the immunoglobulin IgA. This becomes absorbed into the mucous covering the villi surfaces and prevents E. coli and other organisms attaching to the fingers. If they are unable to attach they are unable to cause disease. The secretory IgA also helps to destroy bacteria. After weaning time however no more IgA is available, the levels rapidly decline and bacteria damage the villi causing them to shrink. This atrophy reduces the absorptive capacity of the gut and the ability of the pig to use its food. The enzymes produced by the cells of the villi are likewise reduced. The changes result in malabsorption of food and poor digestion with or without the development of scour. The villi normally regenerate within 5 to 7 days after weaning from cells at their base called enterocytes, which multiply and migrate upwards causing the villi to return to their normal length. The rate of multiplication and regeneration is in part an environmental temperature and energy dependent phenomena. If the pig is weaned in an environment below its lower critical temperature (LCT), the rate of regeneration of the villi is reduced and in some cases ceases. (This results in the hairy pig that doesn't grow). Feed intake is a crucial part of the equation.

 

Before weaning the piglet receives milk as a liquid feed at regular intervals. As a result the bacterial flora of the gut, although relatively simple compared with that of a mature pig, is stabilised.

At weaning cessation of milk removes secretory IgA and there is a period of starvation, followed by irregular attempts to eat solid feed. This results in a dynamic disruption of the bacterial flora of the gut which may last for 7 to 10 days before stabilising. This bacterial disruption may also contribute to poor digestion and possibly scour, particularly when high levels of pathogenic strains of E. coli are involved.

Before weaning the piglets led an ordered life, being "called" with their litter mates to suckle and obtain small amounts of milk at regular intervals, sleeping between meals in a warm creep. All this suddenly changes at weaning, the pigs finding themselves in strange surroundings with strange piglets, and only solid feed. Psychological trauma is inevitable and is likely to affect some pigs more than others, resulting in impaired digestibility and lowered resistance to disease. The more this psychological stress can be minimised the better

If poor growth is evident in the first seven days post-weaning the following options or variables need to be considered:

Check that the weights of all pigs at weaning are to the target level.
Check the ages of the pigs at weaning.
Heavier but younger pigs will have a more immature digestive system.
Group the pigs by weight or keep them in their litter groups.
Use a highly digestible and palatable diet and mix and soak this for the first day or two with water.
Use different diets according to body weight and age.
Use open dishes for feeding for the first three days at least, instead of troughs.
Feed small quantities of creep four to five times daily and remove uneaten stale feed.
Provide easy access to fresh clean water.
Use in-feed medication for the first ten days post-weaning.
Check that the environmental temperature is constant and satisfies the pigs requirements particularly in the first four days post-weaning.
Maintain a dry house without draughts.
Reduce any form of stress.
If pigs are housed on slatted floors provide solid comfort boards for them to lie on for the first few days.
Remove the smallest piglets from each pen after 7 to 10 days and place them together in one pen in the same room. Their diet can then be adjusted accordingly.
Key factors that dictate the degree of villus atrophy
Age of the pig at weaning
Weight of the pig at weaning
The environmental temperature and its fluctuations.
Feed intake and availability of feed.
Digestibility of the feed.
Quality of the proteins.
Levels of milk proteins.
Levels of bacterial and viral challenge.
Nutrition
Only minimal amounts of solid food are eaten during the suckling period and very little before 10 days of age.

Key points to maximising feed intake

Pigs at weaning time will eat a warm gruel better than a solid food.
Gruel feeding reduces the degree of villus atrophy and dehydration.
Pigs need to be encouraged to feed in the first 2 to 3 days post-weaning because the maternal discipline of suckling every 40 minutes is lost.
Provide creep feed for the first 72 hours in open dishes 5 to 6 times a day. This will encourage the pigs to eat and avoid over eating. Piglets naturally "root" pellets from the floors rather than a trough. Recently washed metal troughs have unattractive smells.
By experiment place the feeders in the most attractive part of the pen.
A small pellet or crumb will increase intake. Pellet size should be 2mm or less.
Examine the piglets mouths at weaning time to ensure there has been no damage to the gums during teeth removal. Pigs with sore infected gums will not eat.
Use a highly palatable diet.
Creep feeding / options
The term "creep feed" here means the pre-starter diet offered to piglets before and just after weaning until they can be changed to a cheaper starter diet. When sows were loose-housed in farrowing pens the pre-starter had to be placed in a "creep" where the sows could not get to it. Now that sows are farrowed in crates or tethers the creep is placed outside the warm creep area in a cooler part of the pen to keep it fresh but the term "creep feed" is still used.

There are a number of options:

No creep given pre-weaning.
Different creeps given pre and post-weaning.
Mixed creeps given post-weaning.
A high dense diet used pre-weaning and a low one post-weaning.
A low dense diet used pre-weaning and a high one post-weaning
Restricted feed for varying periods of time.
Choice feeding.
By trial and error determine the best methods that produce a healthy rapid growing weaner.
On most farms the best method is to offer very small quantities of fresh creep feed several times a day for the last 7 to 10 days before weaning and to continue this for one to three days after weaning while gradually changing to starter rations.

Nutritional components of a good creep diet

Whilst it is not the purpose of this book to discuss nutrition in detail nevertheless Fig.9-3 shows the effects on growth rate of a simple diet compared to a complex one. A complex diet could consist of the following:

Cooked cereals 38 %, maze oil 11%, milk products 45%, Glucose and sugars 5% plus minerals and vitamins, MJ DE/kg 16.4, Protein 21 to 23%, lysine 1.3 to 1.4%, oil 20%.





Water

At weaning time the pig's diet changes abruptly from milk as its source of nutrients to water and solid feed and it should therefore be given encouragement to drink. Water is best provided in open cube drinkers, poultry drinkers or water bowls for the first 3 to 4 days. If nipple drinkers are the only source of water many pigs may take up to 24 hours before they drink adequate amounts and if the drinkers are not functioning correctly some may never get enough.





Housing

Good weaning accommodation should satisfy the following criteria;

Be easy to clean, disinfect and be dry.
Provide good observation.
Require minimum handling of waste (faeces, slurry) and separate the pig from its faeces.
Have a simple feeding system that is adaptable for the first 3 to 4 days.
Provide a draught free, well insulated environment that satisfies the needs of the piglets.
Segregate one weeks pigs from another, ideally at least 10m apart, with no direct air or faeces contact.
Segregated disease control or segregated early weaning principles should be adopted. (See chapter 3).
In flat decks and mechanically ventilated housing a fail safe and alarm system should be in operation.
Weaner kennels, deep bedded in straw, provide an ideal post-weaning environment in temperate climates.
If slats are used these should preferably be made of plastic and self cleaning.
Temperature requirements
Satisfying these is essential if a pig is to be weaned successfully. (Fig.9-4). These requirements are dependent upon the weight of the pig, the feed intake, the quality of the feed, the floor type and the air flow. Whilst guidelines can be given for any particular age and group of pigs these should never be relied upon totally, but rather the temperature of the building adjusted to the observed requirements of the pig. A pig in its thermo-neutral zone (its comfort zone) lies on its side making little bodily contact with its contemporaries.

 

Temperature requirements of the pig are increased in wet draughty pens, or where there is high air flow. Always measure the temperature at pig level using a maximum minimum thermometer or computer monitoring equipment, placed in a guarded position.

Stocking density

The stocking density for pigs weaned on fully slatted or partly slatted floors is approximately 0.1m2 per 10kg liveweight. This should provide sufficient room for all pigs to lie down in the pen without body contact. No more than 40 pigs should be housed in each pen, ideally less (this can be doubled in deep straw accommodation) with a maximum of 200 pigs per room. Pens of pigs may be split after 10 to 14 days or the smallest pigs removed and placed together. Do not mix different ages of pigs in one room.

Problems of over stocking:

There is a greater risk of disease developing, particularly greasy pig disease, post-weaning diarrhoea, PRRS, SI and EP.
Growth rates are reduced.
Vice increases including ear and tail biting.
Ventilation problems arise.
Ventilation
This is critical in the first 24 hours of weaning. Draughts must be avoided otherwise the pig loses energy and becomes catabolic with a predisposition to the development of disease. (Fig.9-5).




Fig.9-6 highlights the major diseases of the weaned pig and the major contributing factors. Take particular note of the importance of an adverse environment and bad management.

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« Reply #9 on: September 29, 2008, 03:51:02 AM »

Structure and Appearance of the Skin
(450) At birth the skin and subcutaneous tissues account for up to 10% of body weight but by the time the animal has matured this has dropped to around 6%. The boar's skin over the shoulder blade is thickened by a mat of fibrous tissue. This protects the shoulder when fighting occurs.
The structure of the skin consists of three parts; an outer epidermis, which is the scaly surface of the skin, the dermis which is the main thick part and the sub dermis which consists of fat and connective tissue. The clinical appearance of the skin particularly in white breeds can be a useful guide to the health or disease state of the pig. When an examination is carried out the following should be noted.

Colour - In white skinned breeds, this may range from very pale, suggesting anaemia possibly from intestinal haemorrhage or iron deficiency, to red which may be generalised suggesting possible fever or sunburn, or localised or pimple sized suggesting insect bites or mange. Blue/black extremities (ears, feet, tail, snout) may suggest septicaemia e.g. salmonellosis, toxaemia, or circulatory failures.
Eczema - This describes dermatitis, where serum oozes to the surface giving rise to a wet lesion. It is often seen in traumatic lesions to the ears and flanks as a result of vice
Hair growth - If this is excessive it may be related to low environmental temperatures, poor nutrition, or general ill health resulting from diseases such as pneumonia, swine dysentery or mange.
Inflammation - Infection and inflammation of the superficial layers is called epidermitis and in the deeper parts, dermatitis. Epidermitis is seen typically in greasy pig disease and dermatitis is associated with bacterial infections such as staphylococci, streptococci and erysipelas. The areas of inflammation may coalesce into large patches or remain as discrete small areas or pimples.
Jaundice - The skin is a slight to moderate yellow colour but these changes are more easily observed in the mucous membranes in the eye. Jaundice may be associated with the blood parasite Eperythrozoon suis, leptospirosis, or where there is damage to the liver due to toxins such as aflatoxin, migrating ascarid larvae, or poisons such as warfarin.
Necrosis - When there is restriction of blood supply to an area of the skin the surface tissue dies (called necrosis) leaving a dark area. Such changes are often seen on the teats, tails and knees of piglets as a result of trauma and in skin lesions of erysipelas diamonds, where the causal organisms block the tiny blood vessels supplying small areas of the skin.
Pustules or papules - These are small areas of inflammation usually from 1-3mm in size that have red raised centres that may show evidence of pus, dead black tissue or initially appear as small vesicles (see below). They arise after infection with viruses, streptococci or staphylococci, or allergic reactions to the mange mite.
Vesicles - These are blisters containing clear fluid which are small (< 1mm) in the case of PRRS virus infection, or up to 10mm in pox virus infections. Large confluent vesicles occur around the skin horn junctions and the mouth and tongue in the vesicular diseases such as swine vesicular disease, foot-and-mouth disease, or vesicular exanthema in countries where these occur.

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« Reply #10 on: September 29, 2008, 03:54:27 AM »

Recognising a Worm Problem
 (493) This is carried out by collecting faeces samples from different ages of pigs and examining them for the presence of worm eggs. 25g samples should be taken from the following animals:- 5 lean dry sows, 5 lean suckling sows, 5 separate samples from weaner faeces at 12 weeks of age and 5 separate samples from finishing pigs at 90kg. These are then submitted to a laboratory for examinations.
A 2g portion of each sample is washed through a sieve with saturated salt or zinc sulphate solution and a small amount of this liquid containing worm eggs is flooded into a glass chamber of a known size. The top of the chamber is then examined microscopically as the eggs float to the surface and the numbers of the different eggs are counted (Fig.11-6). The levels per g of faeces are then calculated.

Worms of one kind or another are almost always present in commercial pig herds. Low numbers are no problem but large numbers can cause tissue damage with malfunction of the body systems that are damaged and loss of condition. It can be difficult to assess the significance of a parasite burden but the following procedures may be adopted.


Step 1

Assess the body condition, growth rates and clinical symptoms of the group of pigs

Coughing - consider lungworm but only if the environment could give access to earth worms or beetles. Ascarid larvae as they migrate through the lungs can increase the incidence of pneumonia and coughing.
Wasting - round worms, coccidia, kidney worms or Balantidium coli.
Blood in the urine - kidney worms.
Blood in the faeces - coccidiosis, trichuris infection.
Anaemic pigs - stomach worms.

Step 2

Assess the type of environment and the way it could maintain parasites. Look at the ages of the pigs affected.

Step 3

Assess post-mortem and slaughter house information for evidence of parasites in the following organs:

Liver damage / milk spot - ascarids, kidney worms
The kidney - kidney worms
The stomach - stomach worms
The intestine - ascarids, nodular worms
The large bowel - nodular worms, whipworms, balantidia
Muscle - muscle worms
Step 4
Assess the results of the faeces examinations. The egg output each day is variable and the results must be interpreted by assessing all samples, together with the types of worm eggs, their numbers (Fig.11-7) and the clinical picture. The output of eggs also varies in the sow with the stage of reproduction, with increased outputs of eggs during lactation. Never make a diagnosis on egg counts alone. Judge their significance by Steps 1, 2 and 3.



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« Reply #11 on: September 29, 2008, 03:56:55 AM »

Terminology
(530)
Enzootic (= endemic) disease - This means that the disease, or at least the infection causing it, is permanently present in a population. In relation to diseases of pigs the population may be a herd or the herds in a region, a country or a continent. Strictly speaking 'endemic' should only be applied to populations of people (demos meaning people e.g. democracy) and 'enzootic' should be used for animals but in practice the two are interchangeable.
Epizootic (= epidemic) disease. - A disease which spreads, usually fairly quickly, to a large proportion of the pig population. The term 'endemic' is often used instead but strictly speaking "endemic' applies to people not animals.

Erosion - A shallow ulcer.

Exotic disease - A disease which does not occur in the region or country of your pig farm.

Pandemic disease - A disease which is widespread throughout a region or the world.

Ulcer - A localised inflamed surface or gut lesion from which the skin or surface membrane has been removed by trauma, chemicals or infection.

Vesicle - A blister containing clear or yellow-tinged liquid under the superficial layers of the skin or surface membrane of the mouth or gut.

Vesicular diseases - Viral diseases in which vesicles are a prominent feature. The vesicular diseases of pigs are foot-and -mouth disease (FMD), swine vesicular disease (SVD), vesicular exanthema of swine (VES) and vesicular stomatitis (VS)

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« Reply #12 on: September 29, 2008, 04:00:15 AM »

How to Recognise Poisoning
(564) This is never easy because of the myriad of potentially toxic substances likely to be present on a farm, however always look for the most common and obvious first.
A poison may only affect an individual animal within a group because it was the only one inadvertently exposed to it. For example a toxic dose of medicine was administered by mistake.

Similarly most or all the pigs in a pen, a number of pens or a complete building may be affected, indicating a much wider exposure. Finally a complete herd may be affected - invariably associated with a common feed, water source or airborne pollution.

Step 1- Study the history carefully

Consult with your veterinarian

Is the onset rapid - usually within 48 hours? If so, are a number of pigs affected?
Is only a particular age group affected, for example gilts, sows, sucking piglets? If so, what is common to the group?
What routines, medicines, management procedures have been applied to the group recently?
Is a particular area of the farm or a number of pens affected? If so are there any common factors?
Does the appearance of the condition coincide with the introduction of a new batch of feed or feed ingredients, a change in water or other local change?
Step 2 - Clinical signs
List the clinical signs. (See Fig.13-1 and Fig.13-2).
Common features of poisoning include:
Rapid onset - (There are however exceptions depending on the dose level and period of exposure).
A defined group of pigs affected.
A number of pigs with identical clinical signs.
Not a recognisable disease.
Rectal temperatures are usually normal.

Clinical signs of different poisons
A broad outline of how different poisons affect different systems of the pig is given in Fig.13-1.




This can be used to help identify possible causes, particularly if used in conjunction with Fig.13-2. Specific systems of the body may be affected and develop the following signs:

Circulatory system

Anaemia.
Cyanosis (blue discoloration of skin).
Increased respiration.
Jaundice.
Haemorrhage.
Digestive system
Abdominal pain.
Diarrhoea - with or without haemorrhage.
Rectal prolapse.
Salivation.
Vomiting.
General effects
Generalised malaise.
Reduced feed intake or complete inappetence.
Reduced growth.
Locomotor system
Abnormal gait.
Ataxia.
Incoordination.
Lameness.
Nervous system
Blindness.
Excitation.
Fits.
Incoordination.
Spasmodic movements.
Reproductive system
Abnormal oestrus.
Swollen vulva.
Abortion.
Embryo reabsorption.
Failure of fertilisation.
Respiratory system
Coughing.
Difficulty breathing.
Pneumonia (found at post-mortem examination).
Sneezing, nasal discharge.
Skin
Colour.
Haemorrhage.
Irritation.
Vesicles.
Urinary system
Blood in the urine.
Cystitis / pyelonephritis (found at post-mortem examination).
Excess mineral deposits (found at post-mortem examination or in the urine).
Pus.




Step 3 - Post-mortem examinations and records of mortality

Post mortem examinations may assist in differentiating between a specific disease and toxic conditions. Samples from tissues are probably required for further laboratory tests. The number of deaths and whether they are sudden or after a short or prolonged illness may characterise certain poisons.

The information assessed from steps 1 to 3 will raise a suspicion of poisoning.

Step 4 - Identify the possible sources of the poison

List the chemicals on the farm - sprays, pesticides etc.
List the medicines on the farm.
What injections have been given?
Are rodenticides used and available?
Are parasecticides used?
Could any sources of feed be suspected?
Is there evidence of spoiled or mouldy feed or mould in the feed delivery system?
Consider water, bedding and other environmental contaminants.
Are sprays / disinfectants used.
In out-door herds consider plants, water and environmental contaminants.
Do any of the signs fit into Fig.13-1 or Fig.13-2?
Step 5 - Identify the toxin
Use Fig.13-1 to identify the potential toxin or toxins, together with the history and symptoms.

Step 6 - Read about the poison

Refer to the poison in the text and administer treatments in conjunction with veterinarian advice.

Step 7 - Confirm the poison

Refer samples to a lab for confirmation. (Fig.13-3).



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« Reply #13 on: September 29, 2008, 04:06:22 AM »

Nutrition and Disease
(615) This chapter looks at the role nutrition may have on disease syndromes but for more extensive information on nutrition itself the reader is referred to specific books dealing with this (e.g. The Science and Practice of Pig Production. Whittemore CT Longman Group UK Ltd).
A major role for the stockperson on the farm is to judge the interaction between the pig, its age and/or productive cycle against the quality, content and intake of feed. The role of management in this respect has an important influence not only on the levels of disease in the herd but also whether the pig maximises its biological potential.

The essential nutrients include protein and amino acids, energy, essential fatty acids, water, vitamins and minerals. A guide to the normal requirements is shown in Fig.14-1 both by weight of pig and ration type.

If you feel you have a feed related problem study the list of clinical problems associated with nutrition. First identify the problem by symptoms and this will suggest potential nutrient deficiencies or problem areas. You would be advised at this time to consult with your feed supplier because a knowledge of the composition of the diet will then assist in determining more specific areas, for example insufficient energy or lysine for the particular age group of pigs in that environment. Fig.14-3 relates the clinical signs to possible causes where there are deficiencies or excesses of minerals and similarly Fig.14-4 of nutrients and vitamins, although it would be uncommon today to see many of them. Details of individual nutrients are given in subsequent pages. The most common problems on farms today however relate to the failure of the diet to satisfy the amino acid and energy requirements for the pig.






Clinical Problems Associated with Nutrition

Abortion

Energy
Fungal toxins present
Iron
Anaemia haemorrhage
Aflatoxin
Anticoagulants
Coal tar poisoning
Copper
Iron
Protein
Vitamin E - Selenium (gastric ulcers)
Bone fracture, malformed bones, lameness
Calcium *
Magnesium
Manganese
Nicotinamide
Phosphorus *
Vitamins * - A, D3, E
Diarrhoea, colitis
Excess protein, tapioca
High levels of wheat
Increased levels of potassium and magnesium
Iron
Nicotinamide
Pantothenic acid
Haemorrhage
Vitamin K
Nervous symptoms, incoordination, lameness
Biotin
Calcium
Copper
Magnesium
Manganese
Pantothenic acid
Phosphorus
Vitamins * - A, B6, D3, * E
Water *
Poor growth, poor appetite
All aspects of nutrition *
Amino acids * - lysine, arginine, histidine, isoleucine, methionine, cystine, threonine, tryptophan
Energy *
Feeds of poor digestibility *
Iron *
Magnesium
Phosphorus
Potassium
Protein *
Sodium chloride *
Vitamins -* A, B6, B12, * D3, choline, riboflavin, pantothenic acid niacin
Water *
Zinc
Poor litter size
Choline
Energy *
Folic acid
Lysine *
Other essential amino acids
Vitamin E *
Reproductive failure
Choline
Energy *
Folic acid
Iodine
Lysine *
Manganese
Other essential amino acids
Riboflavin
Vitamins - B12, * E
Respiratory diseases
Energy
Protein
Vitamin E
Skin changes
Iron *
Essential fatty acids * - linoleic acid
Nicotinamide
Potassium
Riboflavin
Salt
Zinc
Sudden death
Selenium *
Thiamine
Vitamin E *
Water - salt poisoning
* Likely to occur. Others uncommon or rare






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« Reply #14 on: September 29, 2008, 04:12:29 AM »

The 5 Freedoms
Freedom from hunger and thirst - (721) Ready access to fresh water and a balanced ration which maintains full health and vigour.
Freedom from discomfort - Provision of a suitable environment and a comfortable resting area.
Freedom from pain, injury and disease - Prevention where possible and prompt diagnosis and treatment when injuries or disease occur.
Freedom to express normal behaviour - Provision of sufficient and appropriate space, interest and the company of other pigs.
Freedom from fear and distress - Sympathetic stockmanship, constant environmental conditions and freedom from aggression by other pigs.
These represent the ideal which can rarely be achieved fully within the practical constraints of an efficient pig farm. Nevertheless they provide a comprehensive starting point from which to assess your own pig farm.
Freedom to express normal behaviour

This is the most difficult one to provide on intensive units. It is also the most controversial, beset by strong emotional and anthropomorphic opinions. It implies freedom of movement, so that all pigs should be able to turn round and carry out their normal bodily functions. This is a contentious issue with respect to the confinement of sows in stalls or tether systems. In the UK dry sow stalls (confinement) and tethers will be illegal from 1st January 1999. The other countries of the EU are partly following this lead by the Council Directive 91/630/EEC. This states that no new sow tethers or conversions to sow tethers should be constructed from the 31st December 1995 but those already in use can continue to be used until the year 2005. Council Directive 91/630/EEC is reviewed by the EU in 1997. Individual pig farmers in countries such as Denmark and the Netherlands, who sell pork products into the UK, are removing sow stalls and tethers so that their products will remain acceptable to UK supermarkets.

Changing from stalls and tethers to loose housing replaces one set of welfare problems with another, particularly in relation to individual sow feed rationing and aggression. Good pen or yard design is required combined with skilled stockmanship if freedom from fear, distress, pain and injury are to be achieved. Housing must be such that animals can stand and lie down without difficulty, have a clean place in which to rest and have visual contact with other pigs.

Other aspects which are not spelt out by the 5 freedoms but which may be implied by them are:

The provision of a caring and knowledgeable management team.
The provision of light during the hours of daylight.
The avoidance of unnecessary mutilation.
The provision of emergency arrangements to cover disasters such as fire, the breakdown of mechanical services and the disruption of supplies.
Humane slaughter.
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